Research Article
Volume 12 Issue 11 - 2021
Central Corneal Thickness in Diabetic and Non-Diabetic Patients Attending B. P. Koirala Lions Centre for Ophthalmic Studies

Rupesh Poudel1, Sanjeeb Kumar Mishra2*, Madhu Thapa3, Niraj Dev Joshi4 and Raju Kaiti5

1Optometrist, Tilganga Institute of Ophthalmology, Kathmandu, Nepal
2Lecturer, Department of Ophthalmology, BP Koirala Lions Center for Ophthalmic Studies, Tribhuvan University, Kirtipur, Nepal
3Associate Professor, Department of Ophthalmology, BP Koirala Lions Center for Ophthalmic Studies, Tribhuvan University, Kirtipur, Nepal
4Instructor, Department of Ophthalmology, BP Koirala Lions Center for Ophthalmic Studies, Tribhuvan University, Kirtipur, Nepal
5Consultant Optometrist, Nepal Eye Hospital Lecturer, National Academy of Medical Sciences, Nepal
*Corresponding Author: Sanjeeb Kumar Mishra, Lecturer, Department of Ophthalmology, BP Koirala Lions Center for Ophthalmic Studies, Tribhuvan University, Kirtipur, Nepal.
Received: August 19, 2021; Published: October 28, 2021




Introduction

The term Diabetes is a disease in which the body cannot produce sufficient insulin to adequately control the level of glucose in their blood [1]. IOP is a routine examination in all the subjects. The measurement of IOP is usually done in both diabetics and glaucoma [2]. The commonly accepted range for normal in the general population is 10 to 21 mm of Hg [2].

It has been traditional to classify Diabetes as being type I or type II. Type I is also called insulin dependent, where the body’s immune system destroys the insulin producing beta cells in pancreas. Type II is characterized by insulin resistance and relative insulin deficiency. Any changes in retina due to diabetes are called Diabetic Retinopathy. Diabetic Retinopathy is again divided as Non-Proliferative and Proliferative type [3].

References

  1. Ederer F and Podger MJ. “The Diabetic Retinopathy study research group”. Controlled Clinical Trials (1984).
  2. Cantor Louis., et al. “Introduction and Definitions: Glaucoma”. In: Am. Academy of Ophthal 1999-2000.
  3. Williams textbook of endocrinology (12th edition), Philadelphia: Elsevier/Saunders 1371-1435.
  4. Wild S., et al. “Global Prevalence of diabetes: Estimates for the year 2000 and projections for 2030”. Diabetes Care 27 (2004): 1047-1052.
  5. Emily Y and Chew MD. “Guest Editorial: A Simplified Diabetic Retinopathy Scale”. American Academy of Ophthalmology 9 (2003): 1675.
  6. Quigley H and Broman A. “The number of persons with Glaucoma worldwide by 2010 and 2020”. Poster presentation at the American Glaucoma Society (2006).
  7. Goldmann H. “Un nouveau tonometre applanation”. Bulletin de la Société Botanique de France Ophthalmology 67 (1954): 474-478.
  8. Ehlers N., et al. “Applanation Tonometry and Central Corneal Thickness”. Acta Ophthalmologica 53 (1975): 54.
  9. Johnson M., et al. “Increased corneal thickness simulating elevated intraocular pressure”. Acta Ophthalmologica 96 (1978): 664-665.
  10. Kanski Jack J. “The Diabetic Retinopathy: Clinical Ophthalmology”.
  11. Whitacre MM., et al. “The effect of corneal thickness on applanation tonometry”. American Journal of Ophthalmology 115 (1993): 592-596.
  12. Claramonte PJ., et al. “Variation of central corneal thickness in Diabetic patients as detected by ultrasonic pachymetry”. Archivos de la Sociedad Española de Oftalmología 81 (2006): 523-526.
Citation: Sanjeeb Kumar Mishra., et al. “Central Corneal Thickness in Diabetic and Non-Diabetic Patients Attending B. P. Koirala Lions Centre for Ophthalmic Studies”. EC Ophthalmology 12.11 (2021): 43-50.

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